MTP
So last night I first took the prescribed MTP II product. MTP is a metalothione promotor. Mt is a protein in the gut which binds to heavy metals and inproves metal metabolism. The warnings indicate that at first, taking MTP could cuase nausea, assumedly from the purging of excess heavy metals. Well, so far, they are right. About 2 pm I felt a flu like feeling, nausea, flgiht fever, and like my thought was fuzzy. I was extremely tired by 4, and felt really awful. My head swam a bit, not like the old days, but teetered all the same. slightly blurred vision adn defntely brain fog- that is the feeling like connections are really hard to make.
Well, its 7:30 now and it is begginign to pass. I dont know how interested you are, but since i have never seen another description of how it feels to be on MTP, my life is, for you, an open book.
Here is how Mt affects copper balnces in teh body:
Copper functions along with iron in many metabolic processes.
Most plasma copper is bound to ceruloplasmin. Histapeniacs are low in ceruloplasmin, as are people with Wilson's disease. I have Cp levels signifigantly lower than lab limits. Copper also binds to superoxide dismutase, which scavenges for superoxide free radicals and protects against oxidative damage.
Copper is absorbed in the small intestine and carried by the copper-binding transport protein metallothione through the plasma. In the plasma it is bound to amino acids, primarily histidine, and to serum albumin for transport to the liver. In the liver, copper is bound to an alpha-globulin to form ceruloplasmin. It is then delivered to cells that have ceruloplasmin receptors on their surface.
Copper is primarily excreted by bile into the gastrointestinal tract, although some unabsorbed copper is excreted through the feces. Biliary excretion is responsible for the body's balance of copper.
Now, I test iwth normal copper levels in ratio to Zinc, but once you compute for Cp, you will find that I have too much unbound copper, or free copper. Mt is supposed to bind to the Cu, in a non-reactive form, then escort it to the liver, where it can be processed with alpha globulin to make Cp. so, this is the hope, cleaning the extra Cu and creating enough Cp. Wish me luck.
Well, its 7:30 now and it is begginign to pass. I dont know how interested you are, but since i have never seen another description of how it feels to be on MTP, my life is, for you, an open book.
Here is how Mt affects copper balnces in teh body:
Copper functions along with iron in many metabolic processes.
Most plasma copper is bound to ceruloplasmin. Histapeniacs are low in ceruloplasmin, as are people with Wilson's disease. I have Cp levels signifigantly lower than lab limits. Copper also binds to superoxide dismutase, which scavenges for superoxide free radicals and protects against oxidative damage.
Copper is absorbed in the small intestine and carried by the copper-binding transport protein metallothione through the plasma. In the plasma it is bound to amino acids, primarily histidine, and to serum albumin for transport to the liver. In the liver, copper is bound to an alpha-globulin to form ceruloplasmin. It is then delivered to cells that have ceruloplasmin receptors on their surface.
Copper is primarily excreted by bile into the gastrointestinal tract, although some unabsorbed copper is excreted through the feces. Biliary excretion is responsible for the body's balance of copper.
Now, I test iwth normal copper levels in ratio to Zinc, but once you compute for Cp, you will find that I have too much unbound copper, or free copper. Mt is supposed to bind to the Cu, in a non-reactive form, then escort it to the liver, where it can be processed with alpha globulin to make Cp. so, this is the hope, cleaning the extra Cu and creating enough Cp. Wish me luck.
posted by warnerk | 4:27 PM
1 Comments:
Luck!!!
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